The Defender Podcast: Gigaohm Biological
The Defender Podcast: Gigaohm Biological High Resistance Low Noise Information Brief (1:50)
The Defender Podcast: Gigaohm Biological High Resistance Low Noise Information Brief (1:50)
Not watched this yet. May add comments later. This is a treat J.C. and Mark from Housatonic both at the top of their game.
Mark and JC (2:58)
Wow, I have never seen J.C. so angry and disappointed (Aug 8th episode). He really lays into Sam Harris and tears him a new one. These people refuse to discuss the biology…..they refuse. They are bought and paid for. Someone like J.C. or Kevin will never work in Academia again. They burnt their bridges and put their money where their mouth is. Respect. It is good to find that there is still some integrity out there. Some people who will stand up and do the right thing no matter the personal cost.
Extremely important – Please watch the video and read the article and distribute
This next video by Jonathan Jay Couey at Gigaohm Biological is one of his best and I will be distributing it widely. It references numerous scientific articles and presents a powerful and well argued case. You are probably as sick as me as hearing and reading about covid but it is extremely important that you understand the implications of the science. I will place the links to the scientific articles (and the abstracts) underneath the video.
What is happening now has spiritual and philosophical implications not just biological (health) implications. I am going to discuss the penultimate article that he refers to in some depth and then offer a working hypothesis on what they are doing and what they are hoping to achieve. Please read the conclusion at the end.
The origin of the virus is key to understanding what is happening. Was the virus man-made or was it a natural virus (zoonotic) that jumped species? We can emphatically state with confidence that it had a lab origin. It was put together in a lab and can trigger numerous inflammatory pathways……this is not normal:
For the moment we leave aside the question of who was involved but I want to point to a tweet from someone who is referred to by J.C. and who exposed what was happening very early on (like Jay did) – Harvard to the Big House:
“Any one of these three concepts alone is enough to act as a smoking gun for an artificial engineered origin for COVID-19, the fact that there are three complementary ones should leave no doubt about the novel coronavirus’s provenance.”https://t.co/PiUXpbFQty— Harvard2TheBigHouse (@Harvard2H) August 2, 2021
SPOILER ALERT: SARS-CoV-2 is a circulating vaccine-derived-coronavirus (cVDCV) via the PLA, but borne from work originally done at UNC, the only institution that’s been attempting to design a LAV for SARS and chimeras needed for attenuation…https://t.co/ocNo2Wm3CM— Harvard2TheBigHouse (@Harvard2H) April 1, 2021
Leaving aside the origins narrative I want to discuss the penultimate article that J.C. refers to as it is crucially important and rather frightening.
The penultimate article that Jay discusses is about an experiment with vaccinated and unvaccinated chickens. Chickens carry a virus disease called Marek which probably originated because of the way they are farmed (so close together). Chickens become infected with Marek by inhalation of dust contaminated with virus shed from the feather follicles of infected birds.
In this experiment they put unvaccinated and vaccinated chickens together. The unvaccinated are birds are called by the fancy name immunologically-naïve sentinel birds. My understanding is that they are a control group. I got this definition on sentinels: “These immune system cells trawl through the epithelial tissues that serve as an interface between our bodies and the outside world: skin, lungs, intestines, etc. Known as the ‘sentinels’ of the organism, they capture all foreign or suspect molecules and carry them to the lymph nodes”.
So, I presume that sentinel is just a reference to the “feather follicles” etc that would form the first barrier (sentinel) to the virus. In other words a group of unvaccinated healthy birds with their immune system etc intact and naïve because it is still natural. Now these sentinel birds come from the same commercial stock as the other birds and so their mothers would have also been vaccinated with Rispens technically called CVI988/Rispens MDV (Marek Disease Vaccine) vaccine. I have made my own diagram and placed a quote from the article underneath:
They used different vaccines during the test. HPV= herpesvirus of turkeys.
MDV has been evolving in poultry immunized with leaky anti-disease vaccines since the introduction of the first vaccines in 1970 All MD vaccines are live viruses administered to 18-day-old embryos or immediately after hatch, and vaccinated birds can become infected and shed wild-type virus . Wild-type MD viruses are so-called serotype 1 viruses. First-generation vaccines include a serotype 3 herpesvirus of turkeys called HVT; second-generation vaccines are a combination of HVT and SB-1, a serotype 2 isolate. Third-generation vaccines are based on an attenuated serotype-1 virus isolate CVI998, the so-called Rispens vaccine.
They also do experiments with Maternal derived antibody and conclude:
Our experiments above show that direct vaccination of birds or vaccination of parent hens makes possible the onward transmission of viral strains otherwise too lethal to transmit, and thus that unvaccinated individuals are put at increased risk of severe disease and death.
So, Marek Viral Disease (MDV) is not eradicated by the vaccine. The vaccine is not a sterilizing vaccine it is “leaky” (does that sound familiar?). It forces the pathogen to mutate and become more virulent. It keeps the shedding (vaccinated) chicken alive longer and so it can walk around and infect the unvaccinated. In nature such a virus would kill the host and die out but because we invent a “vaccine” we are able to keep the chicken alive but turn the virus into something super lethal for the unvaccinated. Geert vanden Bossche warned about this and we wrote an article about MDV months ago:
Ode to a Chicken
I managed to download this one from Twitch so that I have a copy if it disappears. Twitch only keeps the old live-streams up for 60 days.
Here follow all the links (resources) with the abstracts:
The spike protein of SARS-CoV-2 induces endothelial inflammation through integrin α5β1 and NF-κB
Vascular endothelial cells (EC) form a critical interface between blood and tissues that maintains whole-body homeostasis. In COVID-19, disruption of the EC barrier results in edema, vascular inflammation, and coagulation, the hallmarks of the severe disease. However, the mechanisms by which EC are dysregulated in COVID-19 are unclear. Here, we show that the spike protein of SARS-CoV-2 alone activates the EC inflammatory phenotype in a manner dependent on integrin α5β1 signaling. Incubation of human umbilical vein EC with whole spike, its receptor-binding domain, or the integrin-binding tripeptide RGD induced the nuclear translocation of NF-κB and enhanced the expression of leukocyte adhesion molecules VCAM1 and ICAM1, the adhesion of peripheral blood leukocytes, and the permeability of the monolayer. Inhibitors of integrin α5β1 activation prevented these effects. We suggest that the spike protein, through its RGD motif in the receptor-binding domain, binds to integrin α5β1 in EC to activate Rho GTPases, eNOS pathways, and the NF-κB gene expression program responsible for vascular leakage and leukocyte infiltration, respectively. These findings uncover a new direct action of SARS-CoV-2 on EC dysfunction and introduce integrin α5β1 as a promising target for treating vascular inflammation in COVID-19.
August 5, 2021
Vaccine-associated enhanced disease: Case definition and guidlines for data collection, and presentation of immunization data
This is a Brighton Collaboration Case Definition of the term “Vaccine Associated Enhanced Disease” to be utilized in the evaluation of adverse events following immunization. The Case Definition was developed by a group of experts convened by the Coalition for Epidemic Preparedness Innovations (CEPI) in the context of active development of vaccines for SARS-CoV-2 vaccines and other emerging pathogens. The case definition format of the Brighton Collaboration was followed to develop a consensus definition and defined levels of certainty, after an exhaustive review of the literature and expert consultation. The document underwent peer review by the Brighton Collaboration Network and by selected Expert Reviewers prior to submission.
Good advicehttps://t.co/7TE6JxUC1L— Moses Eleftheria (@EleftheriaMoses) August 5, 2021
Effectiveness of the BNT162b2 vaccine in preventing COVID-19 in the working age population – first results from a cohort study in Southern Sweden
Results The estimated vaccine effectiveness in preventing infection ≥7 days after second dose was 86% (95% CI 72-94%) but only 42% (95% CI 14-63%) ≥14 days after a single dose. No difference in vaccine effectiveness was observed between females and males. Having a prior positive test was associated with 91% (95% CI 85 to 94%) effectiveness against new infection among the unvaccinated.
Natural infection showed a 91% effectiveness against new infection. versus 86% effectiveness +7 days after second dose of BNT162B2 immunization.
Antibody response patterns in COVID-19 patients with different levels of disease severity in Japan
We analyzed antibody response patterns according to the level of disease severity in patients with novel coronavirus disease 2019 (COVID-19) in Japan. We analyzed 611 serum specimens from 231 patients with COVID-19 (mild, 170; severe, 31; critical, 30). Immunoglobulin M (IgM) and IgG antibodies against nucleocapsid protein (N) and spike 1 protein (S1) were detected by enzyme-linked immunosorbent assays. The peaks of fitting curves for the optical density (OD) values of IgM and IgG antibodies against N appeared simultaneously, while those against S1 were delayed compared with N. The OD values of IgM against N and IgG against both N and S1 were significantly higher in the severe and critical cases than in the mild cases at 11 days after symptom onset. The seroconversion rates of IgG were higher than those of IgM against both N and S1 during the clinical course based on the optimal cut-off values defined in this study. The seroconversion rates of IgG and IgM against N and S1 were higher in the severe and critical cases than in the mild cases. Our findings show that a stronger antibody response occurred in COVID-19 patients with greater disease severity and there were low seroconversion rates of antibodies against N and S1 in the mild cases.
Kinetics of SARS-CoV-2 specific IgM and IgG responses in COVID-19 patients
The emerging COVID-19 caused by SARS-CoV-2 infection poses severe challenges to global public health. Serum antibody testing is becoming one of the critical methods for the diagnosis of COVID-19 patients. We investigated IgM and IgG responses against SARS-CoV-2 nucleocapsid (N) and spike (S) protein after symptom onset in the intensive care unit (ICU) and non-ICU patients. 130 blood samples from 38 COVID-19 patients were collected. The levels of IgM and IgG specific to N and S protein were detected by ELISA. A series of blood samples were collected along the disease course from the same patient, including 11 ICU patients and 27 non-ICU patients for longitudinal analysis. N and S specific IgM and IgG (N-IgM, N-IgG, S-IgM, S-IgG) in non-ICU patients increased after symptom onset. N-IgM and S-IgM in some non-ICU patients reached a peak in the second week, while N-IgG and S-IgG continued to increase in the third week. The combined detection of N and S specific IgM and IgG could identify up to 75% of SARS-CoV-2 infected patients in the first week. S-IgG was significantly higher in non-ICU patients than in ICU patients in the third week. In contrast, N-IgG was significantly higher in ICU patients than in non-ICU patients. The increase of S-IgG positively correlated with the decrease of C-reactive protein (CRP) in non-ICU patients. N and S specific IgM and IgG increased gradually after symptom onset and can be used for detection of SARS-CoV-2 infection. Analysis of the dynamics of S-IgG may help to predict prognosis.
Kinetics of viral load and antibody response in relation to COVID-19 severity
Abstract Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the causative agent for coronavirus 2019 (COVID-19) pneumonia. Little is known about the kinetics, tissue distribution, cross-reactivity, and neutralization antibody response in patients with COVID-19. Two groups of patients with RT-PCR–confirmed COVID-19 were enrolled in this study: 12 severely ill patients in intensive care units who needed mechanical ventilation and 11 mildly ill patients in isolation wards. Serial clinical samples were collected for laboratory detection. Results showed that most of the severely ill patients had viral shedding in a variety of tissues for 20–40 days after onset of disease (8/12, 66.7%), while the majority of mildly ill patients had viral shedding restricted to the respiratory tract and had no detectable virus RNA 10 days after onset (9/11, 81.8%). Mildly ill patients showed significantly lower IgM response compared with that of the severe group. IgG responses were detected in most patients in both the severe and mild groups at 9 days after onset, and remained at a high level throughout the study. Antibodies cross-reactive to SARS-CoV and SARS-CoV-2 were detected in patients with COVID-19 but not in patients with MERS. High levels of neutralizing antibodies were induced after about 10 days after onset in both severely and mildly ill patients which were higher in the severe group. SARS-CoV-2 pseudotype neutralization test and focus reduction neutralization test with authentic virus showed consistent results. Sera from patients with COVID-19 inhibited SARS-CoV-2 entry. Sera from convalescent patients with SARS or Middle East respiratory syndrome (MERS) did not. Anti–SARS-CoV-2 S and N IgG levels exhibited a moderate correlation with neutralization titers in patients’ plasma. This study improves our understanding of immune response in humans after SARS-CoV-2 infection.
T cell-mediated immune response to respiratory coronaviruses
Emerging respiratory coronaviruses such as the severe acute respiratory syndrome coronavirus (SARS-CoV) and Middle East respiratory syndrome coronavirus (MERS-CoV) pose potential biological threats to humans. SARS and MERS are manifested as severe atypical pneumonia associated with high morbidity and mortality in humans. The majority of studies carried out in SARS-CoV-infected humans and animals attribute a dysregulated/exuberant innate response as a leading contributor to SARS-CoV-mediated pathology. A decade after the 2002–2003 SARS epidemic, we do not have any approved preventive or therapeutic agents available in case of re-emergence of SARS-CoV or other related viruses. A strong neutralizing antibody response generated against the spike (S) glycoprotein of SARS-CoV is completely protective in the susceptible host. However, neutralizing antibody titers and the memory B cell response are short lived in SARS-recovered patients and the antibody will target primary homologous strain. Interestingly, the acute phase of SARS in humans is associated with a severe reduction in the number of T cells in the blood. Surprisingly, only a limited number of studies have explored the role of the T cell-mediated adaptive immune response in respiratory coronavirus pathogenesis. In this review, we discuss the role of anti-virus CD4 and CD8 T cells during respiratory coronavirus infections with a special emphasis on emerging coronaviruses.
Mapping the human genetic architecture of COVID-19
The genetic makeup of an individual contributes to susceptibility and response to viral infection. While environmental, clinical and social factors play a role in exposure to SARS-CoV-2 and COVID-19 disease severity1,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. We describe the results of three genome-wide association meta-analyses comprised of up to 49,562 COVID-19 patients from 46 studies across 19 countries. We reported 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3–7. They also represent potentially actionable mechanisms in response to infection. Mendelian Randomization analyses support a causal role for smoking and body mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19, with unprecedented speed, was made possible by the community of human genetic researchers coming together to prioritize sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease.
Virological and serological kinetics of SARS-CoV-2 Delta variant vaccine-breakthrough infections: a multi-center cohort study
Objectives Highly effective vaccines against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) have been developed but variants of concerns (VOCs) with mutations in the spike protein are worrisome, especially B.1.617.2 (Delta) which has rapidly spread across the world. We aim to study if vaccination alters virological and serological kinetics in breakthrough infections. Methods We conducted a multi-centre retrospective cohort study of patients in Singapore who had received a licensed mRNA vaccine and been admitted to hospital with B.1.617.2 SARS-CoV-2 infection. We compared the clinical features, virological and serological kinetics (anti-nucleocapsid, anti-spike and surrogate virus neutralization titres) between fully vaccinated and unvaccinated individuals. Results Of 218 individuals with B.1.617.2 infection, 84 had received a mRNA vaccine of which 71 were fully vaccinated, 130 were unvaccinated and 4 received a non-mRNA. Despite significantly older age in the vaccine breakthrough group, the odds of severe COVID-19 requiring oxygen supplementation was significantly lower following vaccination (adjusted odds ratio 0.07 95%CI: 0.015-0.335, p=0.001). PCR cycle threshold (Ct) values were similar between both vaccinated and unvaccinated groups at diagnosis, but viral loads decreased faster in vaccinated individuals. Early, robust boosting of anti-spike protein antibodies was observed in vaccinated patients, however, these titers were significantly lower against B.1.617.2 as compared with the wildtype vaccine strain. Conclusion The mRNA vaccines are highly effective at preventing symptomatic and severe COVID-19 associated with B.1.617.2 infection. Vaccination is associated with faster decline in viral RNA load and a robust serological response. Vaccination remains a key strategy for control of COVID-19 pandemic.
Imperfect Vaccination Can Enhance the Transmission of Highly Virulent Pathogens
Could some vaccines drive the evolution of more virulent pathogens? Conventional wisdom is that natural selection will remove highly lethal pathogens if host death greatly reduces transmission. Vaccines that keep hosts alive but still allow transmission could thus allow very virulent strains to circulate in a population. Here we show experimentally that immunization of chickens against Marek's disease virus enhances the fitness of more virulent strains, making it possible for hyperpathogenic strains to transmit. Immunity elicited by direct vaccination or by maternal vaccination prolongs host survival but does not prevent infection, viral replication or transmission, thus extending the infectious periods of strains otherwise too lethal to persist. Our data show that anti-disease vaccines that do not prevent transmission can create conditions that promote the emergence of pathogen strains that cause more severe disease in unvaccinated hosts.
Monitor for COVID-19 vaccine resistance evolution during clinical trials
Although less common than the evolution of antimicrobial drug resistance, vaccine resistance can and has evolved. How likely is it that COVID-19 vaccines currently in development will be undermined by viral evolution? We argue that this can be determined by repurposing samples that are already being collected as part of clinical trials. Such information would be useful for prioritizing investment among candidate vaccines and maximizing the potential long-term impact of COVID-19 vaccines.
One thing you should not do is vaccinate with a non-sterilizing “leaky” vaccine during a pandemic. You should not vaccinate people that have no symptoms and you should especially not vaccinate kids. If you do that you make the virus more virulent. You make it worse. That is exactly what they are doing. Why are they doing it? Are they stupid? Of course not, they know exactly what they are doing. They are destroying natural God given immunity and replacing it with a needle. This will give them the power of life and death at the end of a needle. And because the “gene therapy” is patented they will literally own you.
You will own nothing but they will own you. According to Gordon Brown ex UK PM leaders at the G7 ‘will decide who lives and who dies, who is vaccinated and who is not’
Former PM Gordon Brown says leaders at the G7 ‘will decide who lives and who dies, who is vaccinated and who is not’ and adds ‘you don’t have to make a choice’ between vaccinating those overseas and young people in the UK.#KayBurley https://t.co/18TN2tpRvE pic.twitter.com/ucFwNk7Agd— Sky News (@SkyNews) June 7, 2021
Not only that but once they go fully digital on the block chain with the Central Banks they can regulate how and what you spend. With AI and the surveillance state they can make your digital tokens expire within a week. You don’t spend it, and poof…. it is gone. No such thing as saving. You want to buy some fags and booze or pizza? Oops your digital cash does not work. You want to run away from your digital prison? Oops your digital token refuses to work outside of a 5 km radius (and you are tracked anyway). You said a naughty word about Zuckerburg…oops your social credit score tanks….no food for you tomorrow.
The future is full spectrum dominance of the “citizen”. Your only escape will be death. They will control whether you live or breathe.
The title of this article comes from Rev 18 which describes the fall of Babylon the “Great city” she (The Great Harlot who rides the beast) committed fornication with the Kings of the earth and seduced them with her sorceries (pharmakeia). In her was found not only the wealth of the whole earth but the slaves and the souls (breath or life) of men.
The Great Harlot has replaced God as the giver and taker of life. She has dethroned God from his place among the cherubim (the living creatures representing the double helix wheels within wheels with the flashing Chatoyance (cats- eye Apatite ) phosphor backbone supporting the four bases (faces) of the living creatures (Ezekiel1). She will make her supplicants trans-human. She owns it all and wants it all. Her hubris has reached to the heavens.
Wheels within Wheels pic.twitter.com/qkMNq04gYE— Moses Eleftheria (@EleftheriaMoses) August 5, 2021
This is all planned. They release a bio-weapon and then introduce a “vaccine”. Both Covid and the vaccine are dangerous. Long-term the vaccine spike protein will activate neuro-degeneration or other inflammatory pathways. That should weed out those with weak genetic dispositions. The ones who survive will then be hooked on the needle. Better be a good boy or girl because no jab and you are done. You will only have corporate immunity. Any new disease and a vaccine will be rolled out. What could possibly go wrong? Ever wondered why God made us all different? Why nature likes variety vs mono-culture? In the right circumstances people with a genetic defect might be the only ones to survive a pandemic. These people are really very stupid.
This is your future if you keep vaccinating. Stop vaccinating NOW.
Warning pic.twitter.com/ITWg4LDVjn— Moses Eleftheria (@EleftheriaMoses) August 5, 2021