Spike Protein Destroys Mitochondria

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Spike Protein Destroys Mitochondria

Covid is not just “a flu” as some suggest and the vaccine is not a cure it is a toxic transfection which codes for the spike protein.  This article is part of an ongoing series about mitochondria and lipids because I have a special interest as my family carries the Fabry mutation causing  Lysosomal Storage Disorders (LSD).  Fabry disease modifies mitochondrial structure in four different organs.  Fabry disease, an X-linked genetic condition, results from alpha-galactosidase deficiency and increased accumulation of glycosphingolipids in cardiovascular tissues.  The other blogs in this series can be found here:

https://www.biblaridion.info/blog/my-big-toe/

https://www.biblaridion.info/blog/lsd-and-covid-part-1/

https://www.biblaridion.info/blog/lsd-and-covid-part-2/

So this subject is of specific interest to me. At the moment my feet are fine. No sensations or numbness, no swelling, no red toes and no stabbing pains. My hypothesis is that every-time I encounter the virus it triggers the latent Fabry (I only have an 11% loading) so my strategy is to strengthen overall immune health and particularly mitochondrial health.  I just completed a 24 hour fast (without taking any supplements but drinking percolated coffee) and feel great but I need to lose more weight.  I have been taking Lipoic Acid (ALA and RLA) , CoQ10, Niacin as well as other supplements (Skullcap, D with K2 MK7, quercetin,NAC, C and Zn and the occasional melatonin etc etc). As well as that I am making and drinking my own kefir water, kombucha, kefir milk, making natto and trying to walk every day (got 2 hours in today). I am gardening and pickling my own veg and my chooks are laying eggs.  The Powers That Be have effectively pulled the pin on bio-warfare and we need to stay as healthy as possible.

Spike Protein Destroys Mitochondria (Studies)

It seems that my coffee and fasting are really doing the trick:

Caffeine and Autophagy To Reduce Fats in Liver

Lipids

One of the comments under Walters article was particularly interesting.

Both ozone and UVB (Ultra Violet B) cause lipid peroxidation – the main difference being the amount of antioxidants and ROS produced.

Reactive oxygen species (ROS) are natural byproducts of cellular oxidative metabolism and play important roles in the modulation of cell survival, cell death, differentiation, cell signaling, and inflammation-related factor production. Mitochondria are an important source of ROS (reactive oxygen species) within most mammalian cells. This ROS production contributes to mitochondrial damage in a range of pathologies and is also important in redox signalling from the organelle to the rest of the cell

“UVB irradiation induces lipid peroxidation and reduces antioxidant enzyme activities”

“Ozone causes lipid peroxidation but little antioxidant depletion”

What conditions do we have at altitude – high UVB and low oxygen – those are the environmental conditions produced by the spike protein! Why are athletes more at risk of sudden death? – because their highly metabolic cardiac cells/mitochondria are under so much stress from both the spike protein and the exercise, they release a ton of UV (biophotons) and ROS/free radicals and blow the casket (heart). And all the medications found to be effective in covid have a great capacity to absorb UV light and therefore reduce this “Stress” eg Quercetin

“Inhibition of UVA and UVB radiation-induced lipid oxidation by quercetin”

It is this primordial REDOX balance in life/nature, between UVB and Oxygen that controls the actions of an aerobic cell – evolution has simply allowed adaptation on a multicellular scale.

This is not as fanciful as we have already seen the benefit of light on metabolic health:

Light and Life

Further Research

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Fenofibrate protects lipoproteins from lipid peroxidation: synergistic interaction with alpha-tocopherol

https://pubmed.ncbi.nlm.nih.gov/10380122/

Liothyronine (T3 thyroid hormone) seems to help upregulate mitochondrial biogenesis while decreasing Reactive Oxidative Species ROS, both in patients with mtDNA mutations and normal people. Effect of thyroid hormone on mitochondrial properties and oxidative stress in cells from patients with mtDNA defects

https://journals.physiology.org/doi/full/10.1152/ajpcell.00415.2007

Lipid oxidation that is, and is not, inhibited by vitamin E: Consideration about physiological functions of vitamin E

https://pubmed.ncbi.nlm.nih.gov/34481937/
https://www.hsph.harvard.edu/nutritionsource/vitamin-e/
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Side note to self:   Get some vitamin E and eat more almonds